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| There are a number of other variables or risk factors associated with the development of cerebral palsy. They include infant thyroid hormone levels, coagulation disorders or clotting defects in the mother or infant, vaginal bleeding, hypocalcaemia, polycythaemia, hypoglycaemia in infants, the mother being exposed to radiation or methylmercury and delivery room depression. Over 400 premature babies were discovered to have lower than normal levels of thyroxine and a correlated higher risk of cerebral palsy. Thyroxine is thyroid hormone. This hormone in low levels this is called hypothyroxinemia. It has not been concluded whether this is a sign of the condition or a cause. Infants weighing 1500 g issuing from a mother exhibiting vaginal bleeding at the time of admission were significantly more likely to evolve cerebral palsy if they were more than 29 weeks into gestation. In Britian is has been reported that premature infants with hypoglycaemia were increasingly likely to acquire cerebral palsy as their blood sugar decreased. If a mother is undergoing radiotherapy, CP may be engendered by the intervention. Radian exposure in other contexts can have the same result. There have been several cases where localized environmental damage has resulted in fetus catamination, for example methylmercury poisoning, and the child contracting cerebral palsy. Stanton, Marion, (2002) The Cerebral Palsy Handbook. Vermillion Stanley, Fiona, Blair, Eve, Alberman, Eva. (2000) Cerebral Palsies: Epidemiology & Causal Pathways. Mac Keith Press • |
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